Robot | Path | Permission |
GoogleBot | / | ✔ |
BingBot | / | ✔ |
BaiduSpider | / | ✔ |
YandexBot | / | ✔ |
Title | UCSD |
Description | Computational Cancer Analysis Resources Publications Team Contact Us Computational Cancer Analysis Laboratory at The Center for Novel Therapeutics and Moores Cancer Center We are a tea |
Keywords | N/A |
WebSite | ucsdccal.org |
Host IP | 185.199.108.153 |
Location | - |
Site | Rank |
US$882,004
Last updated: 2022-08-22 22:39:08
ucsdccal.org has Semrush global rank of 12,000,292. ucsdccal.org has an estimated worth of US$ 882,004, based on its estimated Ads revenue. ucsdccal.org receives approximately 101,770 unique visitors each day. Its web server is located in -, with IP address 185.199.108.153. According to SiteAdvisor, ucsdccal.org is safe to visit. |
Purchase/Sale Value | US$882,004 |
Daily Ads Revenue | US$815 |
Monthly Ads Revenue | US$24,425 |
Yearly Ads Revenue | US$293,097 |
Daily Unique Visitors | 6,785 |
Note: All traffic and earnings values are estimates. |
Host | Type | TTL | Data |
ucsdccal.org. | A | 3599 | IP: 185.199.108.153 |
ucsdccal.org. | A | 3599 | IP: 185.199.110.153 |
ucsdccal.org. | A | 3599 | IP: 185.199.111.153 |
ucsdccal.org. | A | 3599 | IP: 185.199.109.153 |
ucsdccal.org. | NS | 21600 | NS Record: ns-cloud-e4.googledomains.com. |
ucsdccal.org. | NS | 21600 | NS Record: ns-cloud-e2.googledomains.com. |
ucsdccal.org. | NS | 21600 | NS Record: ns-cloud-e3.googledomains.com. |
ucsdccal.org. | NS | 21600 | NS Record: ns-cloud-e1.googledomains.com. |
Resources Publications Team Contact Us Computational Cancer Analysis Laboratory at The Center for Novel Therapeutics and Moores Cancer Center We are a team of Cancer Researchers and Computational Biologists working on Cancer Classification, Target Discovery and Precision Oncology. Projects and Collaborations Below are our many ongoing projects and collaborations around our general framework to functionally delineate oncogenic states. These are understood as a defined set of specific and coordinated patterns of aberrant activation/deregulation of signaling pathways, and downstream effectors and transcriptional programs, including the tumor stroma and immune microenvironment, which result in similar phenotypic characteristics and cell vulnerabilities to genetic or pharmacological perturbations. Following this functional approach we use computational and experimental methodologies to define oncogenic states and provide a more accurate biological context, and molecular phenotypes, to |
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